BMS-F
Chemical compound / From Wikipedia, the free encyclopedia
BMS-F is a chemical from the aminoalkylindole family invented by Bristol-Myers Squibb around 1999,[1] that acts as a potent and selective agonist for the cannabinoid receptor CB2, with a Ki of 8 nM at CB2 and 500x selectivity over the related CB1 receptor. It has antiinflammatory effects and inhibits release of TNF-α.[2][3]
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Formula | C27H33N3O5 |
Molar mass | 479.577 g·mol−1 |
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